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Mohamed Anwar Bin-Umer
Mohamed Anwar Bin-Umer
Verified email at rutgers.edu
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Elimination of damaged mitochondria through mitophagy reduces mitochondrial oxidative stress and increases tolerance to trichothecenes
MA Bin-Umer, JE McLaughlin, MS Butterly, S McCormick, NE Tumer
Proceedings of the national academy of sciences 111 (32), 11798-11803, 2014
1082014
Trichothecene mycotoxins inhibit mitochondrial translation—Implication for the mechanism of toxicity
MA Bin-Umer, JE McLaughlin, D Basu, S McCormick, NE Tumer
Toxins 3 (12), 1484-1501, 2011
852011
A genome-wide screen in Saccharomyces cerevisiae reveals a critical role for the mitochondria in the toxicity of a trichothecene mycotoxin
JE McLaughlin, MA Bin-Umer, A Tortora, N Mendez, S McCormick, ...
Proceedings of the National Academy of Sciences 106 (51), 21883-21888, 2009
682009
A Lipid Transfer Protein Increases the Glutathione Content and Enhances Arabidopsis Resistance to a Trichothecene Mycotoxin
JE McLaughlin, MA Bin-Umer, T Widiez, D Finn, S McCormick, NE Tumer
PLoS One 10 (6), e0130204, 2015
322015
Disease Resistant Transgenic Plants
NE Tumer, MAB Umer, J McLaughlin
US Patent App. 12/919,613, 2011
22011
Plant genes that confer resistance to trichothecene mycotoxins and fusarium head blight
NE Tumer, JE McLaughlin, MA Bin-Umer
US Patent 9,428,758, 2016
12016
Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum
MAB Umer
Rutgers The State University of New Jersey, School of Graduate Studies, 2014
12014
Two novel non-specific lipid transfer proteins provide enhanced resistance to a trichothecene mycotoxin by reducing oxidative stress.
JE McLaughlin, MA Bin-Umer, S McCormick, NE Tumer
2014
An Arabidopsis non-specific lipid transfer protein provides enhanced resistance to a trichothecene mycotoxin by reducing oxidative stress.
JE McLaughlin, MA Bin-Umer, T Widiez, S McCormick, NE Tumer
2013
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